It is well known that the texture of the prostate is a soft and elastic gland. This characteristic of the prostate, on the one hand, is conducive to achieving the contraction and relaxation of the prostate; On the other hand, it is also conducive to the normal survival of prostate ganglia and capillaries.
This is precisely why the human body's bone nerves and blood vessels are distributed in the porous and soft periosteum, rather than in the dense and hard bone.
Once calcification occurs in the prostate, it will directly cause necrosis and hardening of the prostate gland tissue, loss of its original elasticity, and then worsen the environment in which the prostate ganglia and capillaries are located, ultimately causing damage to the prostate ganglia and capillaries.
Prostate calcification can cause sexual dysfunction
Initial prostate calcification causes damage to the prostate capillaries, which in turn affects the normal expansion and thickening of the prostate capillaries, and does not provide sufficient (physical) compressive and (physical) pulling stimulation to the prostate ganglia.
Without compressive and pulling stimulation of capillaries, the sensitivity of the prostate ganglion cannot be rapidly improved, which is very detrimental to both penis erection and orgasm acquisition.
With the further development of prostate calcification, damage is caused to the prostate ganglia, which in turn affects the normal sensory and conductive function of the prostate ganglia to sexual stimulation.
The sensory function of the prostate ganglion to sexual stimuli decreases, and it is unable to fully sense sexual stimuli from the outside world. On the one hand, it is unable to fully expand the penile artery; On the other hand, it is not possible to fully close the penis veins.
Inadequate expansion of the penile artery can directly affect the increase in blood injection into the cavernous body of the penis, resulting in insufficient congestion of the penis, and ultimately a decrease in the rigidity of the penis's erection; The inability of the penis veins to achieve sufficient contraction will directly affect the decrease in blood output from the cavernous body of the penis, resulting in an accelerated rate of penile congestion and regression, ultimately leading to rapid atrophy of the penis.