Endometrial tuberculosis is a disease that has a serious impact on women. After falling ill, it not only affects sexual desire but also leads to infertility, which must be reasonably prevented.
Endometrial tuberculosis hazards
1. Sexual boredom: In addition to increased vaginal secretions, patients with endometrial tuberculosis also experience symptoms such as low back pain and abdominal pain, resulting in a sharp decrease in female interest.
2. Harmful to the fallopian tubes and pelvis: When acute endometrial tuberculosis is severe, the endometrium is congested, the uterine body is tender, and further development leads to the appearance of uterine muscle tuberculosis, fallopian tube tuberculosis, and pelvic tuberculosis.
3. Cause infertility: because the main pathological changes of endometrial tuberculosis are endometrial congestion and edema, a large amount of tuberculous exudate, and a large number of plasma cell and lymphocytes infiltrated in the stroma, these changes are not conducive to the ascending of sperm and the implantation and development of pregnant eggs, resulting in infertility.
Etiology of endometrial tuberculosis
1. Immunology believes that in women with normal immune function, the local immune system, mainly composed of endometrial cells flowing into the abdominal cavity from the fallopian tubes during menstruation, is killed. In cases of insufficient local immune function or excessive endometrial cells in the retrograde abdominal cavity, immune cells are killed. It is reported that patients with endometrial tuberculosis have a history of lupus erythematosus and other autoimmune diseases, which is twice as high as patients with this disease.
2. Genetic factors: Simpson proposed that first-degree relatives (female) of patients with endometrial tuberculosis were significantly higher than those in the control group, but no specific HLA antigen related to the disease was found.
3. The chemistry of the body cavity epithelium indicates that the ovarian germinal epithelium, pelvic peritoneum, and pleura all originate from the lymphatic and venous discharge of the body cavity epithelium. Repeatedly stimulated by menstrual blood, hormones, or chronic inflammation, they can generate endometrial sample tissue and form endometrial tuberculosis.
4. According to endometrial cultivation, Sampson first proposed that endometrial fragments shed during menstruation enter the abdominal cavity through the fallopian tubes with retrograde menstrual blood flow, and are implanted in the ovaries and adjacent pelvic peritoneum, continuing to grow and spread, developing into endometrial tuberculosis. Women with genital malformations or obstruction often develop endometrial tuberculosis, indicating that menstrual reflux can lead to endometrial cultivation. Endometrial tuberculosis occurs in abdominal incision or perineal incision after delivery, and the surgical personnel bring the endometrium into the incision for iatrogenic cultivation.